When your mind is caught in a loop — replaying a conversation, fighting an urge, scanning a room for signs that people think you’re strange — the natural instinct is to think your way out. Argue with the thought. Collect evidence against it. Reframe it. Clinicians teach this too: dispute the belief, name the distortion, build a more balanced thought.

Awareness Is the First Step in Healing.

That cognitive work has real value when the brain is calm. In the middle of an active loop, though, it often makes things worse. Trying to analyze a looping circuit with the same thinking that drives it is like trying to put out an electrical fire with more electricity. Thinking is not the way out of thinking.

Your struggles aren’t character flaws — they’re brain biology, and our brains can change. The way out of overthinking, rumination and compulsive habit loops runs through parts of the brain that don’t think in words at all.

Accepting new clients throughout Connecticut via telehealth. Book a free 15-minute consultation or Call/Text: (203) 212-8493.

Why thinking can’t solve thinking

Thinking is self-perpetuating. Conceptual thought is only one function of the brain organ, and it runs largely on a set of midline regions called the default mode network: the medial prefrontal cortex and the posterior cingulate cortex. This network carries self-referential thought, the story of who you are across time, and the replaying that becomes rumination.

When you try to think your way through an intrusive thought, an urge or a moment of social panic, you recruit the same network that is running the loop. Here is how the loop usually turns:

  1. An urge, a body sensation or an intrusive thought arises.
  2. The narrating mind comments on it: “Why am I thinking this? Make it stop.”
  3. Frustration, alarm or resistance follows.
  4. That reaction keeps the self-referential network busy, which feeds the original loop — and it starts again.

But the brain does far more than think. Seeing, hearing, feeling the weight of your feet on the floor — these are brain functions too, and they work without narration. The aim is not to stop thinking. Trying to stop thoughts is like trying to stop a tsunami. Thoughts are useful; we need them. We just don’t want them to hijack the entire brain. The skill is switching: moving attention from the thinking networks to the sensing ones, on purpose, and back again.

Two layers of experience: perception and the story on top

Experience arrives in layers. First comes sensory registration: light reaching the eye, sound waves reaching the ear, pressure on the skin. Then, almost at once, the mind adds two more layers — a feeling tone (pleasant, unpleasant or neutral) and a story about what it means:

  • “This twitch means something is wrong with my nervous system.”
  • “This urge is unbearable.”
  • “I’ve been quiet too long. Everyone thinks I’m strange.”

The brain’s threat circuits receive sensory signals within a fraction of a second, so the layers are not separate in the anatomy. They can be separated in experience. Try it with a taste you dislike. You label it “bad.” Now look closely at the bare sensation on the tongue. Without the feeling tone and the label, it simply is what it is.

The Himalayan contemplative traditions mapped this long before brain imaging. Buddhist psychology treats feeling tone as a mental factor distinct from perception itself and from the conceptual elaboration that follows, and it counts mental awareness as a sixth sense — the one that evaluates the other five. Neuroscience has found a similar split.

Two modes of self-awareness in the brain

In a well-known imaging study, Norman Farb and colleagues compared two ways of attending to yourself.1 In the narrative mode, people reflected on their traits and their story, and midline regions, especially the medial prefrontal cortex, did the work. In the experiential mode, people attended to what they were sensing right now, and activity shifted toward a right-sided network: the lateral prefrontal cortex, the insula (the brain’s map of internal body signals), secondary somatosensory cortex and the inferior parietal lobule. In untrained people the insula and the medial prefrontal cortex were tightly coupled — sensation and story fused. After eight weeks of mindfulness training, the two had uncoupled.

What actually locks the loop: the reaction to the reaction

Here is the part most people miss. An urge — call it craving — has its own feeling tone. Then annoyance about the urge arises, and the two get mixed together. That mixture is what locks in the habit. The friction tells the brain the urge is important and demands relief: the compulsion, the checking, another round of overthinking.

An early Buddhist teaching, the Sallatha Sutta, describes this as two arrows.2 The first arrow is the painful sensation. The second is the reaction we add to it. The untrained mind is struck by both. The trained mind feels the first and does not fire the second. Separating the sensation from the reaction loosens the loop.

Affect labeling: shine a light on the reaction

When an urge or a distressing thought strikes, the usual response is resistance: suppress it, distract yourself, criticize yourself for having it. But resistance is a form of attention. Fighting the urge keeps it at the center of the stage.

Instead, put a light on the reaction itself:

  1. Step back from the urge or thought.
  2. Find your reaction to it: annoyance, impatience, dread.
  3. Rest your attention on that reaction, not on the urge.
  4. Say it out loud and hear yourself say it: “I notice I’m annoyed by the urge.”
  5. Repeat it every time the urge returns. Each repetition is a rep.

The noticing itself is neutral. The annoyance is still there, but you are now perceiving it neutrally, the way you might notice the color of a passing car. You stop being the irritation and become the one observing it.

This draws on a line of research called affect labeling. In a 2007 study, Matthew Lieberman and colleagues at UCLA showed people emotionally charged images while scanning their brains.3 When people labeled the feeling in words, the amygdala and other limbic regions responded less to negative images, and activity rose in one region, the right ventrolateral prefrontal cortex. The more that region activated, the less the amygdala did, and the relationship ran through the medial prefrontal cortex. Putting feelings into words acts like a brake.

In that study, people chose labels from a list; they did not speak. Saying the label aloud is my clinical reasoning, not a finding of the study. Turning an observation into speech, and then hearing your own voice, brings in the language and hearing circuits as well. In my experience, the label lands more firmly when you hear it.

A personal example

For a long stretch, I gagged after brushing my teeth. Nothing was in the back of my throat. In the moment just before I rinsed, the gag would start, every single day. When I watched it closely, I found a very subtle moment, like a switch flipping between “I’m not going to gag” and “I am.” That moment is where the awareness has to go. With reps, it mostly stopped. It still comes back once in a while, and when it does, I notice the annoyance — and then I laugh. The worst case is that it happens, and I still brush my teeth. That shift from fighting to noticing is the whole method.

Training the switch: the salience network

Moving between thinking and sensing on demand takes an agile switch. Vinod Menon and Lucina Uddin described the brain’s switch as the salience network, centered on the anterior insula and the dorsal anterior cingulate cortex.4 It detects what matters, inside the body or out in the world, and shifts resources between the brain’s large networks.

NetworkMain hubsWhat it does
Default mode networkMedial prefrontal cortex, posterior cingulate cortex, inferior parietal lobuleSelf-referential thought, autobiographical memory, imagining what others think, rumination
Central executive networkDorsolateral prefrontal cortex, posterior parietal cortexGoal-directed tasks, working memory, deliberate problem-solving
Salience networkAnterior insula, dorsal anterior cingulate cortexFlags what matters and switches between the other networks

Menon and Uddin suggested this framework may help explain disorders of emotion and social thinking. People caught in rumination often describe exactly that: a switch stuck on the inside, pulled back again and again into self-referential thought.

Shamatha: reps for attention

The switch can be trained the way a muscle is trained. In the Himalayan tradition, the practice is called shamatha, or calm abiding: resting attention on one thing without thinking about it, and returning each time the mind wanders. Each round has four steps:

  1. Anchor. Rest attention on a raw sensation: the hum of a fan, the edge of a doorframe, the pressure of your feet on the floor.
  2. Drift. The mind wanders into commentary, planning or memory. This is not failure; it is how brains work.
  3. Notice. You realize you have drifted. That moment of noticing is the switch coming online.
  4. Return. Without judgment, bring attention back to the anchor.

Every cycle of drift, noticing and return is one rep. The goal isn’t to stay in direct perception all the time. The goal is to exercise the switch, because thoughts and emotions capture attention and pull it away.

What changes with practice? Functionally, the narrative and experiential modes can uncouple with training, as Farb’s study showed. Structurally, the picture is mixed. Long-term meditators show thicker cortex in the insula and prefrontal regions than non-meditators,5 but that comparison cannot show cause, and the largest controlled trial to date — two combined randomized trials with 218 people — found no structural brain change after eight weeks of mindfulness training.6 What we can say is that the switch gets faster with practice; whether the brain’s shape changes on the scale of weeks is still an open question.

The frustration trap

The most common way the practice fails is frustration. A thought arrives, you get irritated that your mind wandered, and that irritation is itself another round of narration. Frustration is the obstacle, so the practice trains the ability to meet it: a thought arises, you notice you’re frustrated, and you bring attention back anyway. The aim is not unbroken focus. It is a faster, more neutral return.

Find your strongest sense

Attention does not work the same way through every sense. I learned violin by ear. Other players read the notes on the page and turned them into music at once; I couldn’t. I sat with the orchestra for ten or so rehearsals, made a great many mistakes, and memorized the music by hearing it — and then I played it with feeling. Everyone’s brain works differently.

Some people anchor most easily through sight: a pattern, a gradient of light, the edge of an object. Others anchor through sound, and others through touch, temperature or the position of the body. Notice which sense holds your attention most easily and lean on it in hard moments. Then give your weakest sense some practice of its own.

This is also how the switch helps in social situations. When a distressing moment hits — at a dinner, in a meeting — look at the button on someone’s shirt, or listen to the actual sound of their voice instead of the commentary about what they might be thinking. The key word is look, not think.

Accepting new clients throughout Connecticut via telehealth. Book a free 15-minute consultation or Call/Text: (203) 212-8493.

Medication as a scaffold, not a crutch

Psychiatric medication is often framed in one of two ways: a permanent chemical fix for a built-in defect, or a crutch that masks symptoms. There is a third way to see it: medication as an intentional, temporary scaffold.

When the prefrontal cortex is flooded by stress, scattered firing or relentless rumination, the executive functions lose their grip. Thoughts spin like wheels with no teeth. The right medication gives them teeth, like gears, so you can engage them and do the work. The sequence looks like this:

  1. Scattered firing and rumination make the reps hard to do.
  2. Targeted medication quiets that noise.
  3. Thoughts get traction, and you can direct them.
  4. You do the reps: labeling, anchoring and the six domains below.
  5. Practice-driven change in the brain builds circuits that hold on their own.
  6. Together with your prescriber, a planned taper becomes possible.

Three examples of targeted, non-controlled medication

When one medication is not enough, a combination can target different circuits. Here are three examples, all non-controlled. Skillful Psychiatry prescribes no controlled substances.

MedicationHow it worksWhat it does in practice
Guanfacine
alpha-2A adrenergic agonist
Stimulates alpha-2A receptors on prefrontal dendritic spines, lowering cAMP and closing HCN channels, which strengthens the connectivity of prefrontal networks7Calms scattered, “popcorn” firing in the prefrontal cortex and restores top-down filtering
Atomoxetine
norepinephrine reuptake inhibitor
Blocks the norepinephrine transporter; because the prefrontal cortex has few dopamine transporters, it raises both norepinephrine and dopamine there, without raising dopamine in the brain’s reward centers8Supports the switch, making it easier to step out of the default mode network and direct attention
Vilazodone
serotonin reuptake inhibitor and 5-HT1A partial agonist
Blocks serotonin reuptake and partially activates 5-HT1A receptors, a design intended to speed onset and add an anti-anxiety effect9In my clinical experience, quiets ruminative drive, leaving room to step back and choose one thought to follow

These three illustrate how a combination can target different circuits. They are not a recommended regimen. Every plan is built for one person, and many people need one medication, or none. For more on non-stimulant options for attention, see ADHD Without Stimulants.

Practice builds what medication supports

Medication does not rebuild circuits on its own. It opens a window of stability in which you can do the work. When you practice affect labeling, sensory anchoring and the lifestyle domains inside that window, you drive activity-dependent plasticity: active circuits release growth factors such as BDNF, synapses that fire together strengthen, and connections remodel.

The long-term aim is not open-ended maintenance; it is a brain that sustains the change on its own. Once those pathways are practiced and steady, the scaffold can be reduced in a planned taper. The timing is decided together with your prescriber, and no one should stop a medication on their own.

The six domains of brain health

Lasting change rests on six domains. Every session at Skillful Psychiatry ends by reviewing them; you pick the one that needs the most work and choose one action for the coming stretch.

Your brain is a physical organ — and like any organ, it can heal and change.

1. Sleep

Sleep consolidates what you practiced during the day. A leading hypothesis holds that sleep scales back the overall strength of connections built while awake and keeps the ones that matter. Sleep was also long thought to speed the clearing of waste from the brain,10 but a 2024 mouse study found clearance slows during sleep,11 so that question is now open. What is not in question: without steady sleep, change is harder.

2. Nutrition

Neurotransmitters are built from what you eat. Serotonin, dopamine and norepinephrine are made from amino acids such as tryptophan and tyrosine, with help from cofactors such as vitamin B6, folate, iron and magnesium. Steady blood sugar matters too: when glucose crashes, the body releases stress hormones to correct it, and that surge works against the calm the prefrontal cortex needs.

3. Exercise

Exercise raises the brain’s growth factors, including BDNF. In animal studies, signals released by working muscle reach the brain and switch on that growth program. Movement primes the brain to reorganize during the attention training that follows.

4. Social connection

The nervous system does not regulate in isolation. Safe, attuned connection with other people lowers stress arousal and quiets inflammatory signaling, which leaves the body in a state where top-down control and emotional flexibility come more easily.

5. Medication

Matched to specific circuits rather than to labels, and positioned as a scaffold, as described above.

6. Brain-healthy activities

Change needs direct practice. A simple place to start is five minutes a day, one sense per day, at the same time each day:

  1. Day 1, sight. Rest your eyes on one object: its edges, its color, the way light falls on it.
  2. Day 2, sound. Listen to one sound: a fan, rain or birds outside.
  3. Day 3, touch. Feel your fingertips on a surface, or your feet on the floor.
  4. Day 4, smell. Hold one scent: coffee, soap, the air from an open window.
  5. Day 5, taste. Attend to the bare taste in your mouth, without calling it good or bad.

When a thought or emotion arises, notice it and return to the sensation. Repeat as often as it takes. Over the days, see how long you can stay with the sensation before the first thought arrives, and aim to lengthen that time without getting frustrated.

Your life in your own hands

Psychiatric distress is not a life sentence, and it is not a personal or moral failing. It is what biological circuits do when they run along deeply worn grooves — and grooves can be re-worn.

Psychiatry can feel frightening: it can seem like putting your life into someone else’s hands. My approach is the opposite. When the neurobiology is explained openly, the relationship changes from symptom containment to collaboration. The clinician’s role is not to create dependence on a pill. It is to offer targeted biological support and practical instruction, at a pace you choose, so that you can reshape your own mind.

I am not here to label you. I am here to help your brain change so you can become the person you were meant to be.

Questions people ask

Why can’t I think my way out of overthinking?

Because overthinking runs on the same self-referential network you would use to argue with it. Analysis keeps that network busy. Shifting attention to a direct sensation — something you can see, hear or feel — uses different networks and gives the loop nothing to feed on.

What is affect labeling, and how do I start?

Affect labeling means putting a feeling into words. When an urge or intrusive thought arrives, name your reaction to it, not the urge: “I notice I’m annoyed by the urge.” Say it out loud, hear it, and repeat it each time the urge returns.

How much practice does this take?

Start small: five minutes a day, one sense at a time. The measure of progress is not how long you stay focused but how quickly and calmly you return when your mind wanders.

Will I need medication forever?

Not necessarily. In this approach, medication is a scaffold that makes practice possible. Once new patterns are steady, many people can reduce or stop medication through a planned taper decided together with their prescriber. Never stop a medication on your own.

Can I get this kind of care by telehealth in Connecticut?

Yes. A. Michael Crook, MSN, APRN, PMHNP-BC, a Board-Certified Psychiatric Nurse Practitioner, offers neuroscience-based psychiatric telehealth throughout Connecticut, with evening appointments until midnight and no controlled substances prescribed. A free 15-minute consultation is available. See Services for details, or read more about the wider lens behind this work on Himalayan Wisdom.

References

  1. Farb NAS, Segal ZV, Mayberg H, et al. Attending to the present: mindfulness meditation reveals distinct neural modes of self-reference. Soc Cogn Affect Neurosci. 2007;2(4):313–322.
  2. Sallatha Sutta (The Arrow). Saṃyutta Nikāya 36.6.
  3. Lieberman MD, Eisenberger NI, Crockett MJ, et al. Putting feelings into words: affect labeling disrupts amygdala activity in response to affective stimuli. Psychol Sci. 2007;18(5):421–428.
  4. Menon V, Uddin LQ. Saliency, switching, attention and control: a network model of insula function. Brain Struct Funct. 2010;214(5–6):655–667.
  5. Lazar SW, Kerr CE, Wasserman RH, et al. Meditation experience is associated with increased cortical thickness. Neuroreport. 2005;16(17):1893–1897.
  6. Kral TRA, Davis K, Korponay C, et al. Absence of structural brain changes from mindfulness-based stress reduction: two combined randomized controlled trials. Sci Adv. 2022;8(20):eabk3316.
  7. Wang M, Ramos BP, Paspalas CD, et al. Alpha2A-adrenoceptors strengthen working memory networks by inhibiting cAMP-HCN channel signaling in prefrontal cortex. Cell. 2007;129(2):397–410.
  8. Bymaster FP, Katner JS, Nelson DL, et al. Atomoxetine increases extracellular levels of norepinephrine and dopamine in prefrontal cortex of rat: a potential mechanism for efficacy in attention deficit/hyperactivity disorder. Neuropsychopharmacology. 2002;27(5):699–711.
  9. Pierz KA, Thase ME. A review of vilazodone, serotonin, and major depressive disorder. Prim Care Companion CNS Disord. 2014;16(1):PCC.13r01554.
  10. Xie L, Kang H, Xu Q, et al. Sleep drives metabolite clearance from the adult brain. Science. 2013;342(6156):373–377.
  11. Miao A, Luo T, Hsieh B, et al. Brain clearance is reduced during sleep and anesthesia. Nat Neurosci. 2024;27(6):1046–1050.
A. Michael Crook, MSN, APRN, PMHNP-BC

A. Michael Crook, MSN, APRN, PMHNP-BC

Board-certified Psychiatric Mental Health Nurse Practitioner. Neuroscience-based psychiatric telehealth throughout Connecticut — evaluation, medication management, and supportive therapy, with no controlled substances prescribed.

Book a Free 15-Minute Consultation

Call or Text: (203) 212-8493